ccl4

Changes of immune function in rats of liver damage induced by CCl4

四氯化碳诱导的肝损伤对大鼠免疫机能的影响

Experimental study on liver fibrosis animal model made by injecting CCL4 intraperitoneally

四氯化碳腹腔注射制备肝纤维化模型的实验研究

Group B : CCL4 model group ;

B组：CCL4模型组；

Again it is found that addition of CCL4 almost double the rate .

这里再一次发现，加入CCL4速度几乎成倍增长。

Test method of wooden granular activated carbon adsorption to CCl4 vapor

GB/T17665-1999木质颗粒活性炭对四氯化碳蒸气吸附试验方法

Hepatic MDA value in CCl4 group was higher than the other four groups .

CCl4组肝脏MDA值组显著高于正常对照组及GSE低、中、高3个剂量组。

Change of Portal Vein Pressure in Rat Liver Fibrosis Induced with CCl4

CCl4所致大鼠肝纤维化门静脉压的变化

Syndrome Identification of CCl4 Induced Liver Fibrosis Model Rats Based on Syndrome Detecting from Recipe Used

基于以方测证的四氯化碳所致大鼠肝纤维化模型的证型探讨

CCl4 could also decrease the synthesis of albumin and the plasma membrane fluidity .

离子交联聚合物膜同时受损肝细胞白蛋白分泌及胞质膜流动性下降。

Methods : Abdominal injection of CCl4 was adopted to duplicate rats acute liver injury model .

方法：采用四氯化碳（CCl4）腹腔注射复制急性肝损伤模型。

CCl4 group 's hepatic TG content was significantly higher than normal group and MDG group .

CCl4组的肝脏中TG值高于正常对照组和GSE中剂量组。

Currently the main production process used is wet process with carbon tetrachloride ( CCl4 ) as solvent .

目前国内生产主要采用湿法工艺，即用四氯化碳（CCl4）做溶剂。

UV spectra of their solution in CCl4 were recorded .

记录了各咔唑衍生物在CCl4中不同光照时间的紫外-可见光谱。

Methods CCL4 was injected intraperitoneally or subcutaneously to make rat liver fibrosis model .

方法分别利用CCL4腹腔注射和皮下注射的方法制备大鼠肝纤维化模型。

Conclusion 0.125 % CCl4 mice acute liver injury model was established and could be applied to health food screening .

结论建立了0.125%CCl4腹腔注射制造的小鼠急性肝损伤模型，可应用于保健食品护肝功能的筛选。

Influence of grape seed extract on rat lipid-peroxidation induced by ccl4 .

葡萄籽提取物对四氯化碳诱导大鼠脂质过氧化反应的影响。

Methods : A rat model of liver fibrosis was induced with 40 % CCl4 in peanut oil .

方法：采用40%CCl4花生油溶液制备大鼠肝纤维化模型，观察白背叶根对肝纤维化大鼠血清和肝组织中多种指标的影响。

The mice model of liver fibrosis induced by CCl4 is a simple , easy and cheaper experimental model .

结论：1.CCl4肝纤维化模型是一种简便、易行、价廉的实验模型。

Experimental study on total flavonoids of buckwheat flowers and leaves on mice hepatic injury induced by CCl4

荞麦花叶总黄酮抗四氯化碳肝损害的实验研究

Changes in different stages of hepatic-fibrosis were detected in model rats induced by CCl4 and composite factors .

结论1.CCl4复合因素诱导的模型大鼠出现阶段性肝纤维化改变。

These results indicate that regenerating hepatocytes shows high membrane stability from which the resistance to CCl4 might result .

这些结果提示大鼠离体再生肝细胞膜稳定性增高可能有抗CCl4损伤的作用。

Results After CCl4 injection , hemodynamic indexes in the CCl4 rats had changed markedly .

结果随着CCl4注射时间的延长，实验组大鼠的血液动力学指标发生了显著的变化。

Conclusion : SPC is highly effective in protection and treatment of acute liver damage in mice induced by CCl4 .

结论：SPC对CCl4造成的急性肝损伤有明显的预防和治疗作用。

A system composed of the nearly immiscible liquids H2O and CCL4 has two phases .

由几乎不相容的液体H2O和CCL4组成的体系具有二相。

Methods Forming a hepatic injury model of mouse and rat by using carbon tetrachoride ( CCl4 ) .

方法用四氧化碳（CCl4）建立小鼠、大鼠肝损伤模型。

The model of chronic hepatic injury was established in SD rats by subcutaneous injection of 10 % CCl4 .

取SD大鼠以10%CCl4皮下注射建立慢性肝损伤模型。

The result indicated that Yin Tian granules have protection effect on experimental liver injury caused by CCl4 on mice .

实验结果表明银天颗粒对小鼠CCl4急性肝损伤具有保护作用。

Conclusion Proliferation and apoptosis related proteins are expressed dynamically in different stages of rat liver fibrosis induced by CCl4 .

结论与细胞增生、凋亡的相关蛋白质表达量在肝纤维化发生发展的不同阶段呈动态变化；在肝纤维化形成过程中细胞增生与凋亡受到相关蛋白质的调节。

Conclusion Injecting CCL4 intraperitoneally is better than Injecting subcutaneously for making a liver fibrosis animal model .

结论制备CCL4肝纤维化动物模型腹腔注射优于皮下注射。

The fluorescence quenching of aromatic oxazole derivatives by CCl4 in different solvents was studied .

本工作对唑类化合物在不同溶剂中的荧光被四氯化碳的猝灭问题进行了研究。