complement activation

Clinical study on serum complement activation in patients undergoing cardiopulmonary bypass

体外循环患者血清补体激活及途径的临床研究

Complement activation is the basic pathological reaction in inflammatory diseases .

补体激活造是炎症反应性疾病的病理反应之一。

The In Vitro Inhibitory Effect of Sodium Selenite on Complement Activation

亚硒酸钠对人补体溶血功能的抑制作用

Conclusions Transitory systemic complement activation takes place after acute ischemic stroke .

结论急性缺血性脑血管疾病早期存在短暂的补体激活。

Experimental Investigation of Complement Activation in Cerebral Ischemia-Reperfusion

脑缺血再灌注中补体激活的实验研究

In Vivo Study of the Inhibitory Effect of Sodium Selenite on Mouse Complement Activation

亚硒酸钠在小鼠体内对补体活化的抑制作用

Changes in cell adhesion molecules and composition of complement activation in patients with acute myocardial infarction

急性心肌梗塞病人细胞粘附分子与补体激活成分的动态变化

CONCLUSION : Molecular structure alteration of IgG increased spontaneous complement activation of IVIG .

结论：IgG分子结构变化增强IVIG自发抗补体活性。

Changes of Regulators of Complement Activation in Pancreas of Experimental Acute Necrotizing Pancreatitis

补体调节因子在急性坏死性胰腺炎中变化的实验研究

The changes of composition of complement activation and interleukin-2 receptor in patients with acute myocardial infarction

急性心肌梗死病人血浆补体活化成分与IL-2R水平的变化

The relation between complement activation and myocardial ischemic damage in the patients with actue myocardial infarction

急性心肌梗死病人补体激活与心肌缺血损伤的关系

Influence on the concentration of plasma endotoxin by inhibition of complement activation in traumatic hemorrhagic shock rats

眼镜蛇毒因子抑制补体激活对创伤失血性休克大鼠血浆内毒素含量的影响

The significance of changes in cell adhesion molecules and composition of complement activation in trauma patients with multiple organ failure

创伤后多脏器功能衰竭患者细胞粘附分子和补体激活成分水平的变化及意义

A serine protease component being responsible to the ability of complement activation were also found in this complex .

一个与补体激活能力相关的丝氨酸酯酶成分也被发现存在于这个复合物中。

The changes of leukocyte rheologic characteristics , cell adhesion molecule and complement activation in patients with coronary heart disease

冠心病病人白细胞流变特性和细胞黏附分子及补体活化状态的变化

The role of complement activation in D-galactosamine / lipopolysaccharide-induced acute hepatic necrosis of rat

补体活化在D-氨基半乳糖/内毒素诱导的大鼠实验性肝坏死中的作用

Complement activation in postburn and their effects on exogenous P.aeruginosa infection in severe burned mice

严重烫伤后补体对外源性铜绿假单胞菌感染的影响

Effect of trichosanthin on mast cell degranulation and histamine release and its relationship with complement activation

天花粉蛋白对肥大细胞脱颗粒和组胺释放的影响及其与补体激活的关系

The dynamic changes in postburn complement activation and their effects on the secretory function of macrophages in severely burned mice

严重烫伤后补体活化的动态变化及对巨噬细胞分泌功能的影响

However , complement activation presents a potential threat to host cells , as inappropriate activation may lead to tissue damage in many diseases .

然而，对机体来说，补体也具有潜在的威胁，其经过不适宜的活化后，可以导致机体多种组织的损伤。

As an important molecule of the innate immune system , MBL triggers the lectin pathway of complement activation .

作为天然免疫系统的重要成份，MBL是补体凝集素激活途径的启动因子。

The involvement of v β 3 integrin and complement activation provides a new area for finding therapeutic measure for pulmonary oxygen toxicity .

αvβ3整合素和补体系统的参与为寻找防治肺型氧中毒的有效手段提供了新的思路。

Rocket Immunoelectrophoresis of C_3 and C_3d : A Simple Sensitive Method for Detecting Complement Activation in Plasma

血浆补体C3活化的简捷敏感检测法

Reused HE , which sterilized with peroxide or formalin , decreases its ability of complement activation .

HE膜用过氧乙酸和福尔马林消毒复用可在一定程度降低其激活补体的能力。

Conclusions Different kinds of dialyser membrane have different ability of complement activation : CU > PMMA > HE > PSN .

结论不同种类透析膜激活补体的能力不同，依次为：CU＞PMMA＞HE＞PSN；

Expression of the Dynamic Changes in Postburn Complement Activation and Their Relation with Endogenous Opportunistic Pathogen Infection in Severely Scalded Mice

严重烫伤后补体动态变化与内源性机会菌感染关系的实验研究

Besides a critical role in innate host defence , complement activation contributes to inflammatory and immunological responses in a number of pathological conditions .

除了在宿主防御中起到关键作用之外，补体激活还在许多病理情况下引起炎症反应和免疫应答。

Glucose-induced loss of glycosyl-phosphatidylinositol-anchored membrane regulators of complement activation ( CD59 , CD55 ) by in vitro cultured human umbilical vein endothelial cells

葡萄糖诱导的体外培养人脐静脉内皮细胞膜中葡糖基磷脂酰肌醇锚式补体活化膜调节物（CD59，CD55）的下调

Connected with cell surface , these three regulators inhibit complement activation through suppressing variously key enzyme in the activation of complement and as a result , relieve inflammation reaction .

MCP、DAF、CD59这三种补体调节因子膜连于细胞表面，通过抑制补体激活过程中各种关键酶而达到抑制补体级联反应的目的，从而减轻机体的炎症反应。

Objectives : To investigate the complement activation and level change of C3d bearing immune complex ( C3d IC ) in patients with uraemia pre and post dialysis .

目的：了解尿毒症患者透析前后补体活化与荷C3d免疫复合物（C3d－IC）水平。