活性氧分子

它们通过快速地移位到炎症部位并活化激发灭菌机制，构成了早期的固有免疫反应，其中一种方式就是快速地发生呼吸爆发，产生大量的活性氧分子（ROS）。

They contribute to the early innate response by rapidly migrating to inflamed tissues , where their activation triggers sterilization mechanisms such as rapid production of reactive oxygen species ( ROS ) in what is called the oxidative burst .

维生素C也可以防止红肿发炎，而且它的抗氧化剂成分能中和破坏细胞，过早使你的皮肤衰老的自由基（高度活性氧分子）。

Vitamin C also may fight inflammation , and its antioxidant properties can neutralize the free radicals ( highly reactive oxygen molecules ) that damage cells and can prematurely age your face .

目的探讨活性氧分子在奥沙利铂诱导凋亡相关基因PUMA表达致肠癌细胞凋亡过程中的作用及影响。

Aim To investigate roles of ROS in oxaliplatin-induced PUMA expression and apoptosis in colon cancer cells .

根据富勒烯材料特有的光化学和光物理性质，将普通的氧气通过载有氯化富勒烯-银胺络合物的Al2O3多孔陶瓷，在Xe灯照射下3O2可能转变成单线态活性氧分子1O2。

A normal oxygen molecule when going through a Al_ ( 2 ) O_ ( 3 ) molecular sieve containing fullerene chlorine-argent amine complex could be changed into an active singlet oxygen ~ ( 1 ) O_ ( 2 ) under the irradiation of Xe lamp .

恶臭气体和气溶胶在接触氧化与活性氧分子。

Odorous gases and aerosols oxidize on contact with active oxygen molecules .

我们假定活性氧簇分子参与了脂多糖诱导的肝细胞脂肪堆积。

We hypothesize that reactive oxygen species ( ROS ) mediate lipopolysaccharide ( LPS ) - evoked hepatic lipid accumulation .

这些活性氧/氮族分子在适量的情况下可作为信号分子参与细胞的正常生理过程。

An appropriate amount of reactive oxygen / nitrogen species can act as signal molecules in cell physiological processes .

游离脂肪酸和葡萄糖诱导人主动脉内皮细胞活性氧产生的分子机制研究

The Mechanism of Free Fatty Acid and High Glucose Induced Intracellular Reactive Oxygen Species Production in Human Aortic Endothelial Cells

但过量的活性氧/氮族分子却一方面可直接引起细胞功能障碍，另一方面通过激活其下游信号通路相关分子而影响细胞内基因的转录调控进而影响蛋白质表达异常。

However , excessive reactive oxygen / nitrogen species can directly cause cell dysfunction and affect the transcriptional regulation of genes through the activation of its downstream signaling pathway .

活性氧在生物大分子如DNA、蛋白质等氧化性损伤中起着非常重要的作用。

Active oxygen has an important role in the oxidative damage of biomacromolecules for example DNA , protein and so on .

活性氧对生物大分子的氧化性损伤

Oxidative Damage of Active Oxygen on Biomacromolecules

小胶质细胞属于脑固有免疫细胞，一旦被激活，通过释放致炎因子和活性氧自由基等分子介导神经炎症。

Microglia is a kind of innate immune cell in the brain . Once activated , microglia mediates nerve inflammation through releasing cytokines , pro-inflammatory molecules and reactive oxygen radicals .

抗氧化酶活性的下降从而使过剩的激发能产生的活性氧分子会进一步对生物大分子以及生物膜产生伤害。

The decrease in the activity of active oxygen scavenging enzymes may result in more active oxygen species which would damage the large biomolecule and membrane . 8 .