炎症性肠病
- 名inflammatory bowel disease
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C反应蛋白反映炎症性肠病的活动性
C-Reactive Protein Reflects Activity of Inflammatory Bowel Disease
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目的探讨髓过氧化物酶(MPO)和超氧化物歧化酶(SOD)作为炎症性肠病(IBD)病情活动临床监测指标的价值。
Objective To evaluate the monitoring action of the expression myeloperoxidase ( MPO ) and superoxide dismutase ( SOD ) activity in the active inflammatory bowel disease ( IBD ) .
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炎症性肠病的外周循环和肠内调节性CD4~+CD25~(high)T细胞表达
Peripheral and intestinal regulatory CD4 ~ + CD25 ~ ( high ) T cells in inflammatory bowel disease
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C反应蛋白对炎症性肠病患者活动性的评价
C-reactive Protein Reflects the Severity of Active Inflammatory Bowel Disease
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P选择素与炎症性肠病
P - selectin and Inflammatory bowel disease
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核因子-кB与炎症性肠病
Nuclear factor kappa B and inflammatory bowel disease
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孕烷X受体基因位点与炎症性肠病的易感性相关
The Pregnane X Receptor Locus Is Associated with Susceptibility to Inflammatory Bowel Disease
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研究显示转录因子核因子(NF)-κB与炎症性肠病关系密切。
It has been shown that the transcription factor nuclear factor ( NF ) - KB is closely related to inflammatory bowel diseases .
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溃疡性结肠炎(UC)是一种病因尚未完全阐明的慢性炎症性肠病。
Ulcerative colitis ( UC ) is an idiopathic chronic inflammatory bowel disease ( IBD ) .
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炎症性肠病患者的vWF、血小板检测及其临床意义
Detection of vWF and platelet in the patients of IBD and their clinical implications
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电刺激疑核对炎症性肠病大鼠IL-2、IFN-γ表达的影响
Effect of the Electrical Stimulation of Nucleus Ambiguus on the Expression of IL-2 , IFN - γ in Rats with TNBS-induced Colitis
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炎症性肠病(InflammatoryBowelDisease,IBD),是一组病因不明的慢性肠道炎症性疾病,目前还没有针对性的特殊治疗。
Inflammatory bowel disease ( IBD ) is a group of chronic intestinal inflammatory disease , whose etiology is unknown . There is no curative treatment .
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炎症性肠病(IBD)的病因和发病机制至今仍未完全明确。
The etiological factors and mechanism of inflammatory bowel disease ( IBD ) are not clear yet .
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炎症性肠病(IBD)是一种天然免疫性疾病。
Inflammatory bowel disease ( IBD ) is a kind of the intestinal immune system disorder disease .
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目的通过制作实验性炎症性肠病(InflammatoryBowelDisease,IBD)的动物模型来探讨IBD的发病机制并观察中药灌肠治疗后对其T淋巴细胞及其粘附分子表达的影响。
Objective To discuss the pathogenic mechanism and observe the effect on T lymphocyte adhesion molecule in animal model of inflammatory bowel disease ( IBD ) .
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炎症性肠病动物模型的建立及外源性IL-10和益生菌诱导粘膜免疫耐受治疗炎症性肠病的效应探讨
Investigation on the Construction of Animal Model of Inflammatory Bowel Disease and the Therapeutic Efficacy of Mucosal Tolerance Induced by IL-10 and Probiotic
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炎症性肠病(IBD)具有遗传异质性,许多免疫调制分子基因的突变可导致炎症发生。
Inflammation arises from a number of mutations of immunomodulatory molecules , supporting the concept of genetic heterogeneity for IBD .
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抗补体C5a单克隆抗体对炎症性肠病的治疗作用研究
The Investigation of the Treatment Effect of Anti-C5a Antibody in Inflammatory Bowel Diseases
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目的分析并探讨炎症性肠病(IBD)患者肠外表现的特点和临床意义。
Objective To analyze and examine the characteristics and roles of extra-intestinal manifestations accompanied with inflammatory bowel disease ( IBD ) .
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Syndecan-1与炎症性肠病发病机制关系的初步探讨
A Preliminary Study on the Relationship between Syndecan-1 and Inflammatory Bowel Disease
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目的研究炎症性肠病(IBD)患者血液中血小板计数和平均血小板体积是否可以作为评价IBD活动性的参考指标。
Objective To study role of platelet count and mean platelet volume in evaluation of inflammatory bowel disease ( IBD ) activity .
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炎症性肠病(IBD)病变主要累及肠道,但亦常合并各种肠外表现。
Inflammatory bowel disease ( IBD ) predominantly affects the gastrointestinal system but it is associated with a large number of extraintestinal manifestations .
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IL-23在炎症性肠病中的免疫调节作用
Immunoregulatory effects of IL-23 on inflammatory bowel disease
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目的:探讨低氧诱导因子1α(HIF1α)、诱导型一氧化氮合酶(iNOS)在炎症性肠病(IBD)中的表达及作用。
AIM : To investigate the expression and action of HIF 1 α and iNOS genes in inflammatory bowel diseases .
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结论:与未患炎症性肠病的患者相比,患有炎症性肠病的患者复发性VTE风险增加。
CONCLUSIONS : Patients with IBD are at an increased risk of recurrent VTE compared to patients without IBD .
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炎症性肠病(IBD)、大肠癌是一组主要累及结肠、直肠组织的难治性消化道疾病。
The effects of mucins on the tissues of normal enteron , IBD and colon cancer are being paid close attention by more and more researchers .
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IL-23受体在炎症性肠病患者外周血淋巴细胞中的表达及意义
Expression of IL-23 receptor on peripheral blood lymphocyte in patients with inflammatory bowel disease and its significance
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~(99m)Tc-hIgG在炎症性肠病诊断与随访中的临床应用
Clinical Application of ~ ( 99m ) Tc-hIgG in the Diagnosis and Follow-up of Patients with Inflammatory Bowel Diseases
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方法采用BALB/c小鼠,经直肠注入OXZ制成炎症性肠病(IBD)模型;
Methods BALB / c mice was used , colitis model was induced by intrarectal injection of oxazolone ;
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目前的研究表明免疫异常是炎症性肠病发病的重要因素,此文就OX40/OX40L的生物学功能及其在IBD中的作用作一综述。
Abnormal immune response is one of important factor in the pathogenesis of IBD . This review summarizes the biological function of OX40 / OX40L and their possible role in IBD .