细胞毒作用

结论TNF能成倍提高化学药物的细胞毒作用，且应在先使用化学药物的基础上给药。

Conclusion TNF can remarkably increase the cytotoxicity of antitumor drugs . In clinical therapy , we should give TNF after antitumor drugs administration .

生物学机制，负责的细胞毒作用的ATA尚未得到很好的理解。

The biological mechanism responsible for the cytotoxicity of ATA is not yet well understood .

肿瘤坏死因子α和干扰素α与丝裂霉素C在体外的协同细胞毒作用

Synergistic Enhancement of Cytotoxicity of Mitomycin C on Gastric Cancer Cells in vitro by Tumor Necrosis Factor a and Interferon α

实验提示N-rasRNA降低并不是细胞毒作用而是药物对细胞DNA合成抑制的结果。

The degression of N-ras RNA was not the effect of cytotoxin but of drug inhibition on DNA Synthesis .

结论雷公藤红素通过阻碍DNA合成及细胞毒作用抑制血管内皮细胞的体外增殖。

Conclusion Tripterine inhibits proliferation of vascular endothelial cells in vitro through hindering the synthesis of DNA and toxicity on cells .

方法细胞毒作用以MTT法测定；

Methods Cytotoxicity was determined by tetrazolium ( MTT ) assay .

抗CD3-抗胶质瘤双特异性抗体体外细胞毒作用的多因素分析

Analysis multiple factors of cytotoxicity to glioma cells by anti CD 3 × anti glioma bispecific antibody

方法采用MTT法检测Ara－C的细胞毒作用。

Methods Cytotoxicity of Ara C was detected by the MTT assay .

应用ATP法测定淋巴细胞体外细胞毒作用的实验研究

Experimental studies on the measurement of the in vitro cytotoxicity of lymphocytes by the ATP method

MTT法测定HM与HCPT细胞毒作用的体外研究

Detection of cell toxicity for HM and HCPT using MTT method in vitro

目的：进一步研究茶多酚（TP）在体外细胞毒作用、体内抗肿瘤的作用。

Purpose : To study cytotoxic effect and antitumor effect of tea polyphenoles ( TP ) .

抗EB病毒口服液对EB病毒抗原表达的抑制作用及其细胞毒作用

Inhibition of Anti-EB Virus Liquor on EB virus antigen expression and its cytotoxicity

抗体依赖性细胞毒作用的~（51）Cr释放测定法及对肺结核患者的初步检测

The ~ ( 51 ) cr release assay of antibody-dependent cytotoxicity ( adcc ) and a preliminary observation of ADCC activity in tuberculosis

CEA单抗-MTX结合物的选择性细胞毒作用

Selective cytotoxicity of CEA mcab-methotrexate conjugate

内毒素（endotoxin，ET）有很强的细胞毒作用，引起发热、白细胞骤降及内毒素血症等，最终可因内毒素休克死亡。

Endotoxin has a strong cytotoxic action which can cause fever , rapid leukopenia , sepsis , shock and even death .

目的探讨维生素E与维生素C联合抗石英尘细胞毒作用及其机制。

Objective To explore anti cytotoxiciity effects and mechanism of Vitamin E ( VE ) combined with Vitamin C ( VC ) on quartz exposed alveolar macrophage ( AM ) .

用MTT法检测PAB的细胞毒作用；

Cytotoxicity of PAB was detected by MTT .

选择性COX-2抑制剂联合顺铂对肺癌的协同细胞毒作用及其机制研究

Study on synergistic cytotoxic effects and mechanism of selective cyclooxygenase-2 inhibitor and cisplatin against human lung cancer cell lines

目的探讨β胡萝卜素（βCarrot，βC）抗石英尘细胞毒作用及其机理。

Objective To study the effect and the mechanism of β - carotene on the injured alveolar macrophage ( AM ) exposed to quartz in vitro .

结论与游离药物相比，脂质体包封提高了盐酸拓扑替康的体外细胞毒作用及体内抗肿瘤效果，而经PEG修饰的脂质体相比于普通脂质体体内、外抗肿瘤效果更强。

Moreover , these results also indicate that PEG-modified liposome provide the better cytotoxic effect and anti-tumor effect compared with conventional unmodified liposome .

方法：用台盼蓝拒染法检测抗EB病毒口服液对脐带血B淋巴细胞的细胞毒作用；

Fuel cells METHODS : The cytotoxic effect of anti-EB virus delicious on B lymphocytes from umbilical cord blood was assayed by using the trypan-blue trophochrome method .

为研究紫杉醇脂质体的体外细胞毒作用及体内抗瘤作用。进行噻唑蓝（MTT）体外试验法和腹腔给药体内抗瘤试验。

The cytotoxic effect and antitumor effect of paclitaxel liposome were studied by using MTT assay method in vitro and in vivo antitumor test .

DAF比色试验在检测血清补体依赖性细胞毒作用中的应用

Application of DAF Colorimetric Assay to Detecting Complement - dependent Cytotoxicity in Serum

结果：在一定浓度范围内，HM与HCPT的细胞毒作用与药物浓度均呈线性关系；

Result : There was line relationship between the cell toxicity of HM and HCPT and their drug concentrations ;

方法在不同试验条件下，用同位素标记的方法，检测巨噬细胞介导的细胞毒作用及PKc的活性。

Methods Macrophage mediated cytotoxicity and protein kinase C ( PKc ) activity under different conditions were measured by isotope labelling method .

实验表明，这三种铂络合物都有较强的细胞毒作用，其中顺铂最强而CCP最弱。

The results showed that the order of the cytotoxicity induced by these platinum complexes was DDP JM-8 CCP in CHL cells .

其机理可能与针刺相关穴位改善UC大鼠结肠粘膜的血流量，降低NO对局部粘膜的细胞毒作用，从而对结肠粘膜产生保护作用有关。

The mechanism of the effect may like improving the flowing blood of colon mucosa , reducing the cell toxicity and protecting the colon mucosa .

结果表明，TNF的直接细胞毒作用可致HUVEC转变成CEC的细胞形态，TNF浓度增加可能是病理情况下CEC增多的重要原因。

The results suggested that cytotoxicity of TNF can induce HUVEC to form CEC , which may be important in increasing CEC under certain pathological conditions .

结论Lef对实验性肝纤维化预防作用可能是通过降低NO的水平，进而降低细胞毒作用和纤维化进程而产生的。

CONCLUSION Lef has significantly preventive action on CCl4-induced liver fibrosis possibly mediated by reduction of NO.

结论细小病毒H1的细胞毒作用可能与其影响胃癌细胞MAPK信号传导途径中相关基因的表达有关。

Conclusions The cytotoxic effects of parvovirus H-1 might affect the gene expression involved in MAPK signal transduction pathway in gastric carcinoma HGC27 cells .