醋酸铅

  • 网络Lead acetate;PbAc
醋酸铅醋酸铅
  1. 结论:按Flint评价标准,醋酸铅是脑细胞分化的特异抑制剂,它的脑细胞发育毒性与抑制细胞的增殖和分化有关。

    Conclusion : According to the standard of Flint , PbAc can be a specific inhibitor of brain cell differentiation , its brain cell developmental toxicity is related to the inhibition of cell proliferation and differentiation .

  2. 运用彗星试验检测醋酸铅对小鼠离体、在体生殖细胞的DNA损伤作用

    Study of DNA Damage of Lead Acetate on Germ Cells of Mice in Vivo and in Vitro

  3. 维生素C对醋酸铅所致小鼠血中金属元素变化的干预作用

    Intervention effect of vitamin C on changes of serum metallic element in mice of lead acetate poisoning

  4. 结果:醋酸铅染毒后小鼠睾丸细胞DNA单链断裂,出现彗星状拖尾。

    Result : Lead acetate induces the breakage of DNA single strand in testis and resulted in comet cells with tail .

  5. 醋酸铅试纸法在低产H2S酿酒酵母筛选中的应用

    Application of lead acetate test paper in the screen of low-H_2S-producing yeast strains

  6. 结果表明:醋酸铅作用可引起细胞凋亡基因Bcl-2的表达下调。

    Results show that : lead acetate can cause the expression of cell apoptosis gene Bcl-2 decline .

  7. 目的探讨外源性超氧化物歧化酶(SOD)对醋酸铅免疫毒性的拮抗作用。

    Objective To study the effects of exogenous superoxide dismutase ( SOD ) on antagonizing immunotoxicity induced by lead acetate .

  8. 结论醋酸铅使睾丸TGFβ1Caspase-3的表达增强,从而促使生殖细胞发生凋亡。

    Conclusion Lead acetate increases the expressions of TGF β _1 and Caspase-3 , which induces apoptosis of germ cells .

  9. 醋酸铅对PC-12细胞Bax、Bcl-2和p53蛋白表达的影响

    Influence of lead acetate on the expressions of Bax , Bcl-2 and p53 in PC-12 cells

  10. 方法4周龄Wistar雄性大鼠,按体重用45mg/kg醋酸铅灌胃30天;

    Methods Four weeks old male Wistar rats were exposed to lead by intragastric administration with 45mg / kg .

  11. 目的试比较醋酸铅氢氧化铵法和PH还原糖法(改良班氏法)在婴幼儿粪便乳糖定性检测中的应用。

    Objective The purpose is to compare plumbum acetate and ammonia hydroxide method with the PH and reduction lactose method on application of qualitative lactose measurement in babies ' excrement .

  12. 化学分析的结果表明,加热脱水后的无水醋酸铅中Pb(OAc)_2的含量为99.30wt%。

    Chemical analysis revealed that the concentration of lead acetate in the dehydrated sample was 99.30wt % .

  13. 结论:低浓度醋酸铅可持续激活大鼠海马神经元原代培养细胞胞质PKC,抑制原代神经元细胞生长。

    Conclusion : Low level lead exposure can result in sustained activation of PKC and inhibit neuron growth in dose dependent manner .

  14. 用蛋白免疫印迹法研究醋酸铅对PC-12细胞Bax、Bcl-2和p53蛋白表达的影响。

    The expressions of Bax , Bcl-2 , and p53 in PC-12 cells after exposure to lead acetate were detected by Western blot .

  15. 甲状腺机能减退后再用醋酸铅处理,皮层、海马组织NGF面密度和平均灰度未见明显改变。

    The area density and the average gray value of NGF protein in lead acetate treated hypothyroid rat brain were of no significant changes .

  16. 本文主要对以丁醇钛,无水醋酸铅,和自制乙醇锆为原料制备锆钛酸铅(PZT)微粉及陶瓷的溶胶凝胶(sol-gel)方法进行了研究。

    It was studied that ultrafine powders and ceramics of lead-zirconate-titanate can be prepared by sol-gel technique with titanium butoxide and anhydrous lead acetate and zirconium alkoxide .

  17. 醋酸铅体外诱发人外周血淋巴细胞HPRT基因位点突变的研究

    Study on Frequency of HPRT Locus Mutation in Human Peripheral Lymphocytes Induced by PbAc_2 in vitro

  18. 目的:通过建立亚慢性铅中毒动物模型探讨醋酸铅对雄性小鼠睾丸细胞DNA的损伤,为进一步了解其毒性作用机制提供科学依据。

    Object : To establish the model of subchronic lead exposure in mice , and explore the effect of DNA damage of testicular cells in order to provide the scientific basis for further understanding of the mechanism of testicular toxicity .

  19. 醋酸铅对ST-8814细胞神经丝蛋白、微管结合蛋白mRNA水平的影响

    Effect of lead acetate on mRNA expression of neurofilament protein and microtubule associated protein in ST-8814 cell line

  20. 0.075%醋酸铅染毒组小鼠脑内NO含量、SOD活性明显下降(P<0.01),而MDA含量明显升高(P<0.01)。

    The content of NO and activity of SOD in mice 's brain was lower in 0.075 % group ( P < 0.01 ), but MDA was higher ( P < 0.01 ) .

  21. 采用水热合成技术,以钛酸四丁酯和三水醋酸铅为原料,NaOH为矿化剂,合成了直径约12nm的钙钛矿结构钛酸铅纳米线。

    PbTiO_3 nanowires with perovskite structure are synthesized by hydrothermal method , using lead acetic and tetrabutyl titanate as the starting materials and NaOH served as a mineralizer .

  22. 方法分别给兔腹腔注射不同剂量的醋酸铅或静脉注射不同剂量的氯化镉溶液,于4h和24h后处死动物。

    Methods Rabbits were given different doses of lead or cadmium respectively . 4 and 24 hours after lead or cadmium administration , animals were sacrificed .

  23. 对成年c(57)BL/6雌性小鼠自由饮用含铅0~2000ppm的醋酸铅溶液后进行了体液免疫功能实验。

    Adult female C_ ( 57 ) BL / 6 mice were exposed to lead acetate in the drinking water at concentrations ranging from 0 to 2000 ppm for 3 and 8 weeks , and the immunizing function of the spleen was tested thereafter .

  24. 介绍了3个方面的硫含量检测方法:①测定油品硫含量的主要方法有库仑滴定法、X射线法、醋酸铅法、化学发光法和紫外分光法等;

    Test methods for sulfur content in three aspects were introduced . ( 1 ) The main methods testing sulfur content in oil products are coulometric titration , X-ray fluorescence analysis , lead acetate test , chemiluminescence and ultraviolet spectrophotometry etc.

  25. 方法给3个实验组兔子每公斤体重分别腹腔注射含5、25或50mg的Pb的醋酸铅溶液,对照组注射生理盐水。

    Methods The rabbits in three experimental groups were given intraperitoneal injection of 5,25 or 50 mg Pb / kg body weight and thc control group was given normal saline .

  26. westernblot检测结果显示:BACE-1条带随玻璃体注射醋酸铅浓度的升高而明显增宽、增强,呈剂量依赖性改变。

    Western blot showed that BACE-1 band was becoming broader and stronger with increased concentration of injecting lead acetate to vitreous body , which revealed a dosage dependent manner . 3 .

  27. 神经细胞中Bcl上、Bax和NGFR蛋白表达在低铅、高铅干预组和损伤组间亦有显著差异。【结论】腹腔注射醋酸铅建立动物损伤模型具有简便可靠、易于控制。

    Conclusion The animal damage model of developing brain of neonatal rats with Intraperitoneal injection of acetate Pb solution is of simple , convenient , reliable and controllable .

  28. 在出生时采用腹腔注射醋酸铅溶液建立新生大鼠铅损伤模型,各组剂量分别为0,20mg/kg,60mg/kg。

    The animal model of Pb-exposed neonatal rats was set up with intraperitoneal injection of acetate Pb solution at birth and injection dose respectively use 0 , 20 , 60mg / kg .

  29. 将新生Wistar大鼠按腹腔注射醋酸铅的剂量,随机分为正常、低铅组、高铅组。各组注射剂量分别为0,20,60mg/kg。

    Neonatal Wistar rats were divided into the control group , low Pb group and high Pb group , and Pb 3 ( AC ) 2 at doses of 0,20,60 mg / kg and respectively .

  30. 本文研究了碱式醋酸铅(BLA)对化学致癌物诱发小鼠肺肿瘤的影响。

    The effects of basic lead acetate ( BLA ) on the induction of lung tumors were studied in mice treated with chemical carcinogens .