淋巴结炎

lín bā jie yán
  • lymphadenitis;bubo;adenitis
淋巴结炎淋巴结炎
  1. CT误诊组织细胞坏死性淋巴结炎1例

    Histiocytic necrotizing lymphadenitis misdiagnosed by CT : case report

  2. 颈部结核性淋巴结炎CT表现

    Cervical Tuberculous Lymphadenitis : CT Manifestations

  3. 腮腺淋巴结炎继发化脓性腮腺炎的CT及声像图特点

    The characteristics of CT and ultrasonography of acute suppurative parotitis secondary to lymphadenitis

  4. 羊干酪样淋巴结炎PCR诊断方法的建立及其比较研究

    Establishment of PCR Diagnostic Method of CLA and Comparative Studies

  5. 淋巴结炎性增生和肿瘤转移动物模型的建立及其MRI初步研究

    Establishment and MRI Study of Animal Model of Hyperplastic and Metastatic Lymph Node

  6. 方法回顾性分析二例猫抓病性结肠淋巴结炎的临床及CT表现。

    Methods The clinical data and CT features of colonic lymphadenitis in two cases of CSD were analyzed retrospectively .

  7. 结论结合病史,CT扫描对猫抓病性结肠淋巴结炎的诊断有很大价值。

    Conclusion combination of the cat contaction history , CT scanning is of great value in the cat scratch disease .

  8. 目的提高对猫抓病性结肠淋巴结炎CT表现的认识。

    Objective To make a further understanding of CT manifestations of colonic lymphadenitis in the cat scratch disease ( CSD ) .

  9. 肠系膜淋巴结炎中血清IgA的表达及临床意义

    Expression of serum IgA in mesenteric lymphadenitis and its clinical significance

  10. 结论:颈部结核性淋巴结炎CT表现为密度不均,环状强化及多发结节融合伴周围脂肪间隙闭塞。

    Conclusion : CT manifestations of cervical tuberculous lymphadenitis are : in homogeneous density of nodes , rim enhancement , nodes mixed together as masses and surrounding fat space disappeared .

  11. 对照组为10例慢性淋巴结炎、3例TNHL和2例HD。

    10 chronic lymphadenitis , 3 T NHL and 2 HD were selected as control group .

  12. 目的探讨血液中血清IgA含量的变化对肠系膜淋巴结炎诊断的意义。

    Objective To investigate the sense of the content of serum IgA in blood about the diagnosis of mesenteric lymphadenitis .

  13. 结果:颈部结核性淋巴结炎CT表现:(1)密度不均多见,约72.22%(13/18),呈规则环状强化或环状强化伴有壁结节;

    Results : The CT manifestations of cervical tuberculous lymphadenitis were : 1.The density of nodes were inhomogeneous in 72.22 % ( 13 / 18 ), rim enhancement and / or with wall nodes ;

  14. 1例反应性淋巴结炎和20例正常人均未检测到IgH和TCRγ基因重排。

    There were no clonal gene rearrangements detected in 1 case of reactive follicular lymphadenosis and 20 normal subjects .

  15. Kikuchi淋巴结炎病理变化规律与发病机制研究

    Regularity of Pathologic Changes and Pathogenesis of Kikuchi-Fujimoto Lymphadenitis

  16. 目的探讨Kikuchi淋巴结炎的病理组织学特点,病理诊断及鉴别诊断。

    Objective To investigate the pathologic-histology characters , pathologic diagnosis and distinguish diagnosis of Kikuchi lymphadenitis .

  17. 方法我们采用改良抗酸染色法(IK)和聚合酶链反应(PCR)法对30例病理学诊断为非特异性淋巴结炎和15例正常胎儿石蜡包埋组织重新切片进行检测分析。

    Method : Paraffin embeded tissue from 30 cases of pathologically diagnosed non-specific lymphadenitis and 15 normal embryo were resectioned and examined with both methods of IK and PCR .

  18. 目的研究人微小病毒B19感染与组织细胞性坏死性淋巴结炎(HistiocyticNecrotizingLymphadenitis,HNL)的关系。

    Purpose To investigate if parvovirus B19 infection play a role in the pathogenesis of true histiocytic necrotizing lymphadenitis ( HNL ) .

  19. 方法对48例颈部淋巴结结核(A组)和32例非结核性淋巴结炎(B组)患者分别进行超声及临床触诊检查,将其结果与病理作对照,并用χ2检验进行统计学处理。

    Methods 48 patients with cervical tuberculous lymphadenitis ( group A ) and 32 patient with non tuberculous lymphadenitis ( group B ) were examined by ultrasonography and palpation . The results were correlated with pathologic findings and χ 2 test was used for statistical analysis .

  20. 目的:探讨组织细胞坏死性淋巴结炎(HNL)的诊断和治疗。

    Objective : To discuss the diagnosis and treatment of histiocytic necrotizing lymphadenitis ( HNL ) .

  21. 其他为皮炎或口炎、血小板减少性紫癜、腹泻病、川畸病、肠系膜或颈淋巴结炎、肾炎或肾病和CNS感染等。

    Other disease induced by EBV infection included dermatitis or stomatitides , thrombocytopenic purpura , diarrhea disease , Kawasaki disease , mesentery or cervical adenitis , nephritis or nephropathy , CNS infection , etc.

  22. 结果:淋巴结炎、B细胞淋巴瘤组和T细胞淋巴瘤组中Survivin阳性表达率分别为:46.67%、73.33%、85.00%,差异有统计学意义(P<0.05);

    Results : Expression rate of Survivin was 46.67 % , 73.33 % , and 85.00 % in lymphadenitis , B cell lymphoma and T cell lymphoma tissue , respectively , and there was significant difference ( P < 0.05 ) .

  23. 目的通过对9例小儿组织细胞坏死性淋巴结炎(HNL)进行总结分析,探讨其诊断和治疗方法。

    Objective To summary 9 cases of histiocytic necrotizing lymphadenitis ( HNL ) of children and discuss the diagnosis and therapy .

  24. 目的:组织细胞坏死性淋巴结炎(HistiocyticNecrotizingLymphadenitis,HNL)是一个误诊率极高的少见病,近年来开始引起临床医生的重视,对它的认识逐渐提高。

    Objective : Histiocytic necrotizing lymphadenitis ( HNL ) is a highly misdiagnosed and rare disease which is paid much more attention to by the clinicians in recent years .

  25. 方法应用免疫组化法S-P法检测43例非霍奇金淋巴瘤组织(其中初治病例32例,复发病例11例)和10例坏死增生性淋巴结炎中VEGF、Bcl-2及P-gp。

    Methods : VEGF , Bcl-2 , P-gp were tested by immunohistochemical method SP on tissues of 43 cases ( including 32 newly diagnosed cases , 11 relapsed cases ) of NHL patients and of 10 cases of narcotizing hyperplasic lymphadenitis .

  26. ACTH与5-HT阳性标志物在2组淋巴结炎组织中分布大致相似,主要位于副皮质区的T淋巴细胞、淋巴滤泡生发中心细胞及巨噬细胞等部位,但表达强度有显著性差异(P<0.05)。

    ACTH and 5-HT positive markers in the lymphadenitis tissue of the two groups showed similar distribution , mainly in cortex of lymph gland T lymphocytes and lymph follicle germinal center cellulae and macrophages etc , but expressive intensity had significant difference ( P < 0.05 ) .

  27. 目的探讨Kikuchi淋巴结炎病理形态变化规律及与非霍奇金淋巴瘤(包括NKT细胞淋巴瘤)、不典型分枝杆菌结核、猫抓病鉴别的诊断。

    Objective To investigate the characteristics of pathological morphology and differential diagnosis of histiocytic necrotizing lymphadenitis ( HNL ) and its distinction with other cervical lymphadenopathy , such as non-Hodgkin lymphoma , atypical Mycobacterium tuberculosis and cat scratch disease .

  28. 结论MMP9、TIMP1高表达可能在结核性淋巴结炎发病机制中发挥重要作用,MMP9/TIMP1失衡可能预示疾病进展和结核播散。

    Conclusion The expression level of MMP-9 and TIMP-1 protein may play an important role in tuberculosis destruction mechanisms and the higher ratio of MMP-9 / TIMP-1 ( the imbalance between MMPs and TIMPs ) may indicate a higher risk of progression of disease and dissemination of tuberculosis .

  29. 小儿亚急性坏死性淋巴结炎9例报告

    A report of 9 cases with subacute necrotic lymphadenitis of child

  30. 外科手术治疗快速进展结核性颈淋巴结炎

    Treatment of Rapidly Rapid Progressing Tuberculous Cervical Lymph Nodes by Surgery