atra
- 网络全反式维甲酸;维甲酸;全反式视黄酸;甲酸;急性早幼粒细胞白血病
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Results There were obvious differences in gene expression between human lung cancer cells before and after ATRA treatment .
结果在全反式维甲酸诱导前后的细胞之间存在明显的基因表达差异。
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Differentiation-Inducing Effect of ATRA on Leukemia Cell Line K562
全反式维甲酸对K562细胞的诱导分化作用
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No significant cells apoptosis induced by ATRA . Difference was significant .
透射电镜观察结果表明,ATRA对该细胞无明显凋亡诱导作用。
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The effects of ATRA on immune response induced by gene immunization
全反式维甲酸对基因免疫应答的调节作用
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Objective ATRA can restrain proliferation and promote differentiation in various tumor cells .
目的全反式维甲酸(ATRA)对肿瘤细胞具有广泛的抑制增殖、促进分化作用。
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But ATRA had no positive effect on DHEA function .
而全反式维甲酸对DHEA的这一作用并无明显增强作用。
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Study of IFN α effect on HL-60 cells or ATRA resistant HL-60 cells
干扰素对HL-60细胞与维甲酸耐药HL-60细胞作用的研究
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We tried to modify the traditional ATRA induction way to increase the efficiency of differentiation .
我们尝试对传统的ATRA诱导法加以改进,提高其诱导分化的效率,建立一种相对简便、高效和可信的诱导方法。
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Neither As 2O 3 nor ATRA showed direct inhibition effect on telomerase activity .
As2O3及ATRA对端粒酶活性均无直接的抑制作用。
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Conclusion There are significant improvement in prognosis of APL treated with ATRA .
结论全反式维甲酸对急性早幼粒细胞性白血病患者的预后有很大改善。
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In addition , ATRA induced ICAM-1 protein expression in vivo and in vitro .
ATRA在体内外诱导ICAM-1蛋白表达。
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Results ATRA mixed micelles injection is stable .
结果维A酸磷脂混合胶团注射剂常温避光放置稳定。
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Conclusions : The differentiation of HL-60 cells can be induced by ATRA .
结论:ATRA能诱导HL-60细胞分化;
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Study on Mechanism of hyperleukocytosis induced by ATRA in patients with APL
APL经ATRA诱导后高白细胞征机制的进一步研究
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Effect of ATRA on Mesangial Cell Strain Proliferation and Type ⅳ Collagen in SD Rat
全反式维甲酸对SD大鼠系膜细胞株增殖和Ⅳ型胶原的影响
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Effect of ATRA and VD_3 on chemosensitivity of primary lung cancer cells
全反式维甲酸联合维生素D3对肺癌原代细胞化疗敏感性的影响
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Conclusions : atRA could promote the differentiation of rabbit BMSCs into neural cells in vitro .
结论:全反式视黄酸能够在体外促进兔骨髓间充质干细胞向神经细胞分化。
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We offered the theory evidence of combining using ATRA and HSV-TK / GCV gene system .
为联合应用ATRA和HSV-TK/GCV基因系统治疗膀胱癌提供理论依据。
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Conclusion : The ATRA liposome was homogeneous and stable , with high encapsulation efficiency .
结论维甲酸脂质体包封率高,粒径小而均匀,稳定性好。
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Activities of anticoagulation and fibrinolysis turned gradually to normal after treatment with ATRA .
经ATRA治疗后各项指标逐渐恢复正常。
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Effects of ATRA on DNA synthesis and expression of TGF β mRNA in vascular smooth muscle cells
全反式维甲酸对血管平滑肌细胞增殖和转化生长因子-βmRNA表达的影响
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APL patients were treated with ATRA for induction remission .
ATRA诱导缓解治疗急性早幼粒细胞白血病202例。
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Cells were collected when induced by ATRA for 48 hours , 96 hours and 144 hours .
加入药物进行干预后,分别于48h、96h和144h收集各组细胞。
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Objective To study the mechanism of drug resistance of APL to ATRA and the methods of reversion .
目的揭示急性早幼粒细胞白血病(APL)产生维甲酸耐药的机制,探讨耐药性逆转的方法。
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Molecular Mechanism and Signal Pathway of TPA / ATRA in Regulation of Cell Cycle in Gastric Cancer Cells
TPA/ATRA调控胃癌细胞周期的分子机制及其信号调控途径
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Determination of all-trans-retinoic acid ( ATRA ) soft gelatin capsule by HPLC
高效液相色谱法测定全反式维甲酸软胶囊的含量
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ATRA also plays a certain role in promoting differentiation of RAR α 2 + myeloma cells .
ATRA对RARα2+骨髓瘤细胞的分化也有一定促进作用。
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BMP-2 , ATRA and RVD affected the differentiation of neural stem cells significantly ;
BMP-2、ATRA和RVD(ATRA+VD3+DEX的简称,余类推)对神经干细胞分化具明显诱导作用;
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Effect of Fraction H of Naja naja atra Venom on Experimental Atherosclerosis
中华眼镜蛇毒组分H对实验性动脉粥样硬化的影响
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Identification of HL 60 cell differentiation related proteins induced by atRA using nano-ESI-MS / MS
纳升电喷雾质谱鉴定维甲酸诱导HL60细胞分化相关蛋白质