烧伤后心功能障碍
烧伤后心功能障碍-
而抑制烫伤后心室肌及肺组织的Gi2α和Gi3α基因的转录可能是可乐定、三七皂苷降低烧伤后Giα表达及其抗烧伤后心、肺功能障碍的重要因素。
The inhibitory effects of Clonidine and PNS on the Gi2 α and Gi3 α gene transcription was one of the important mechanisms of the reduction of Gi α expression , which subsequently lead to the alleviation of cardiac and pulmonary dysfunction after scald .