醛固酮

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  • aldosterone
醛固酮醛固酮
  1. 肾上腺皮质的主要产物为醛固酮、皮质醇和脱氢异雄酮硫酸盐。

    The principal adrenocortical products are aldosterone , cortisol and dehydroepiandrosterone sulphate .

  2. 醛固酮诱导心肌重构以及天然维生素E对其干预的影响

    Myocardial Remodeling Induced by Aldosterone and Inhibited Partially by Natural Vitamin E

  3. 原发醛固酮增多症腺癌的CT诊断

    CT Diagnosis of Aldosterone - Producing Adenoma

  4. 单纯性肥胖儿童血浆血管紧张素II醛固酮对心脏结构的影响

    Effects of renin-angiotensin system on myocardial hypertrophy in simple adipose children

  5. 血管紧张素Ⅱ和醛固酮对心脏成纤维细胞Ⅲ型胶原mRNA表达的影响

    Influence of angiotensin ⅱ and aldosterone on type - ⅲ collagen mRNA expression in cultured cardiac fibroblasts

  6. COPD和肺心病急性期患者肾素血管紧张素醛固酮系统的变化

    Renin-Angiotensin II-Aldosterone System Changes at Acute Stage of COPD and Cor Pulmonale

  7. 与手术病理比较,B超在醛固酮腺瘤和双侧肾上腺增生中的诊断符合率分为95.77%及73.33%;CT为98.51%及31.03%;

    The coincidence of ultrasonography was 95.77 % and 73.33 % in APA and BAH , 98.51 % and 31.03 % with CT .

  8. 现已证实,肾素-血管紧张素-醛固酮系统(RAS)在心室重构中起非常重要的作用。

    RAS plays an important role in LV remodeling after AMI .

  9. 阻断血管紧张素I向血管紧张素II的转化,防止肾上腺皮质分泌醛固酮。

    Block the conversion of ANG I to ANG II and prevent the secretion of aldosterone from the adrenal cortex .

  10. RAS阻断剂治疗原发性高血压醛固酮逃逸现象临床观察

    Aldosterone escape in treatment of essential hypertension with RAS blockers

  11. 本文对我院从1990年5月到1994年3月间经手术病理证实的23例原发性醛固酮增多症的肾上腺CT表现作了回顾性分析。

    The adrenal CT manifestation of 23 cases of primary hyperaldosteronism , all proved by surgery and pathology , from 1990 to 1994 were analysed .

  12. 目的:探讨CT及体位刺激试验对原发性醛固酮增多症(原醛)分型、定侧诊断的价值。

    Objective To evaluate the value of CT and posture stimulation test ( PST ) in the differential diagnosis and lateralization of primary hyperaldosteronism ( PA ) .

  13. ESWL对肾素-血管紧张素-醛固酮系统和血压的影响

    Effects of ESWL on renin - angiotensin-aldosterone system and blood pressure

  14. 结论ET与肾功能、血压及紧素一血管紧张素一醛固酮系统关系密切。

    Conclusions The increase of plasma ET 1 may have an intimate relation to renal function and renin angiotensin aldosterone system .

  15. 结果:与C组比较,术后2月H组收缩期左心室压力下降速率(-dp/dt)显著降低;左心室重量、心肌血管紧张素和醛固酮含量显著增高;

    Results : Two months later , - dp / dt decreased significantly , while left ventricular mass , plasma level of angiotensin ⅱ and aldosterone in H group increased significantly .

  16. 螺内酯、氯沙坦对AMI大鼠血浆胶原代谢产物及心肌组织心钠素、醛固酮的影响

    Impact of spironolactone , losartan on the plasma collagen metabolism and cardiac atrial natriuretic peptide , aldosterone levels in AMI rats

  17. ANF和ACTH对体外培养的人正常肾上腺组织分泌醛固酮的影响

    Effects of ANF and ACTH on Aldosterone Secretion in Cultured Human Adrenal Tissue

  18. 目的:观察醛固酮拮抗剂&螺内酯对原发性高血压(EH)患者心肌纤维化的影响。

    Objective : To explore the effects of aldosterone antagonism spironolactone on cardiac fibrosis in patients with essential hypertension .

  19. 方法于ACEI治疗开始前和治疗后1、3、6个月分别测定30例心衰患者卧位醛固酮、血管紧张素II的血浆浓度,分析其变化规律。

    Methods The concentration of aldosterone and angiotensin ⅱ was measured before and after 1,3 and 6 months treatment of ACEI .

  20. β-受体阻滞剂、血管紧张素转换酶抑制剂与血管紧张素受体拮抗剂及醛固酮受体拮抗剂可降低DCM心力衰竭的死亡率。

    Betablocker , ACE inhibitor , angiotensin receptor blocker and aldosterone blocker are efficient in reducing mortality .

  21. PKC被证明在多种类型细胞上醛固酮诱导的信号通路中是一种重要的中介物。

    In several cell types , PKC has been reported to be an important mediator in aldosterone-induced signaling pathway .

  22. 用细胞培养、内皮素放射免疫测定和RTPCR的方法,探讨醛固酮对心室成纤维细胞分泌内皮素的影响。

    Using cell culture , radioimmunoassay for endothelin and RT PCR , the effect of aldosterone on the endothelin secretion of ventricular fibroblasts was studied .

  23. 结论:ACE基因多态性与ACE密切相关,而与血清血管紧张素Ⅱ及醛固酮没有关系。

    Conclusion : The ACE gene polymorphism is closely interrelated with ACE without reference to the serum AT ⅱ and ALD .

  24. 联合应用ACEI和醛固酮受体拮抗剂能更有效的改善高血压患者的动脉弹性。

    Combination ACEI with aldosterone antagonists improved aorta elasticity in patients better with essential hypertension .

  25. 充血性心衰ACEI治疗中醛固酮逃逸现象与心率变异的研究

    Aldosterone Escape and Heart Rate Variability in Patients with Congestive Heart Failure After ACEI Treatment

  26. 结果位于转录调节区-334位点的醛固酮合酶基因型分为TT、TC及CC型三种;

    Results Three genotypes for aldosterone synthase at position 334 in the transcriptional regulatory region were found : TT , TC and CC .

  27. 利用放射免疫法测定肾素、血管紧张素Ⅱ(AngⅡ)及醛固酮的浓度,紫外法测定血管紧张素转换酶(ACE)活性。

    Concentration of renin , angiotensin II , aldosterone , and activity of angiotensin I converting enzyme ( ACE ) were measured respectively .

  28. 所有病例术前均经B超、CT扫描、血管造影及取肾上腺静脉血测醛固酮浓度4种定位方法检查。12例不明原因的高血压行肾血管造影及血浆肾素水平的测定。

    CT scanning ; Angiography ; Adrenal venous sampling to measure the concentration of aldosterone . Angiography and selective venous sampling for renin in renovascular hypertension were performed for 12 cases , with one of hematuria .

  29. 其中肾素&血管紧张素系统(RAS)、影响肾钠处理如细胞骨架蛋白以及醛固酮分泌的基因被认为是原发性高血压重要的侯选基因。

    The genes encoding RAS , influencing renal sodium handling such as adducing and secretion of aldosterone are considered as the important candidate genes .

  30. 两组治疗后血浆肾素(PRA)、血管紧张素(AngⅡ)、醛固酮(ALD)水平较治疗前均下降(P<0.05)。

    Renin , angiotensin ⅱ and aldosterone in plasma in two groups were significantly decreased ( P < 0.05 ) .