甲基汞

甲基汞致小鼠肝细胞DNA的损伤

A study on DNA damage of mouse livers induced by methylmercury

甲基汞、电离辐射对小鼠胸腺DNA合成及适应性反应的影响

Effects of Methylmercury ( MeHg ) and Ionizing Radiation on Adaptation and DNA Synthesis of Thymus of Mice

GC法测定地表水饮用水中的甲基汞

Detection of methylmercury in surface water and drinking water by GC method

实验结果表明：甲基汞可使中国林蛙的DNA甲基化率有不同程度的降低。

The experimental results show that : methylmercury can make the DNA methylation rates of Rana chensinensis reduce at different levels .

甲基维生素B（12）、维生素B（12）降低甲基汞含量及免疫与行为毒性作用的研究

Effect of methylcobalamin and vitamin B_ ( 12 ) on the contents of methylmercury , immune toxicity and behavioral teratogenesis caused by methylmercury in rats

甲基汞对大鼠脑即刻早期基因c-junmRNA表达影响

Effect of Methylmercury on Expression of Immediate Early Gene c-jun mRNA in Rat Brain

氯化甲基汞在ITO电极上的电化学行为及其降解

The Electric Chemical Behavior and Degradation of Methyl-mercuric Chloride at ITO Electrode

丰富运动环境下甲基汞对海马神经毒性减轻效果的morriswatermaze实验评价

The Estimate of the Effect of Environmental Enrichment Lightening the Methylmercury Toxicity to Hippocampal Gyrus by Morris Water maze Test

氯化甲基汞诱导C6胶质瘤细胞凋亡及其机制的研究

The Mechanism of Methylmercury Chloride Induced-apoptosis in C6 Rat Glioma Cell

甲基汞对脑神经胶质细胞凋亡及c-fos表达的影响

Effect of methylmercuric chloride on apoptosis and c-fos expression of brain glial cells

甲基汞诱导大鼠大脑皮层c-JUN蛋白表达

Induction of methyl mercury on c-JUN protein expression of rat cerebral cortex

硒与甲基汞对体外培养细胞SCE的作用

Effect of selenium and methylmercury on SCE in cells cultured in vitro

氯化甲基汞对APL模型裸鼠的作用研究

Effect of Methyl Mercury Chloride on Nude Mice of APL Model

甲基汞对小鼠精原细胞SCE的影响

Influence of Methylmercury on SCE of Spermatogonia of Mice

甲基汞对大鼠大脑皮层即刻早期基因c-FOS蛋白表达影响

The Effect of Methyl Mercury on the Expression of Immediate Early Gene c-FOS Protein in Rat Cerebral Cortex

目的：探讨氯化甲基汞（MMC）对发育脑组织损伤的机理。

? Objective : To study the mechanism of Chloride methylmercury ( MMC ) damage of the developing brains .

氯化甲基汞染毒对亲仔两代ICR小鼠脑、肝、肾中汞分布的影响

Distribution of Mercury in Various Organs of Maternal Mice and Offspring after Maternal Mice Treated with Methylmercury

采用体外全胚胎培养模型和显微镜检查方法，对接触不同剂量氯化甲基汞的离体SD大鼠胚胎进行了病理学观察。

Morphological observations of teratogenicity of methylmercury chloride ( MMC ) on 9 5 day isolated Sprague Dawley rat embryos were studied with whole embryo culture in vitro and optical microscopy .

方法采用原位末端标记（TUNEL）和免疫组织化学方法研究甲基汞对小鼠睾丸生殖凋亡及其凋亡基因的表达。

Methods TUNEL assay and immunohistochemistry assay were used to examine the apoptosis effect and the expression of apoptosis genes of germ cells in male mice .

目的探讨低剂量氯化甲基汞（methyl-mercurychloride，MMC）对小鼠胚胎肠上皮细胞凋亡的影响及相关机制。

Objective To study the effects and related mechanism of low-dosed methyl-mercury chloride ( MMC ) on the epithelial apoptosis of fetal mice intestine .

目的对SD大鼠经腹腔注射氯化甲基汞（MeHgCl）的急性染毒方式，观察其对大鼠脑组织和血清某些抗氧化指标的变化。

Objective To study the effects of methylmercury chloride ( MeHgCl ) on some antioxidant indexes in rats .

目的：观察低剂量氯化甲基汞（methylmercurychloride，MMC）对胎鼠肠上皮细胞凋亡及凋亡相关蛋白表达的影响。

Objective : To study the effect of methylmercury chloride ( MMC ) on the apoptosis and the expression of apoptosis-associated proteins in the intestinal epithelium of fetal mice .

结果接触氯化甲基汞生后3d的大鼠脑细胞核3H－dAMP的掺入量明显低于相应未接触氯化甲基汞的对照组（P＜0.05）。

The amount of 3H dAMP incorporation into the nuclei of rats from exposed group of 3 day old was significantly lower than that of the unexposed group of 3 day old ( P < 0 05 ) .

结果表明：甲基汞浓度为0.1μg／ml时，神经管上皮层细胞脱落，室管膜层变薄和排列无序；尿生殖窦、肝脏等亦出现一定病理变化。

The results showed : when dose of MMC was 0 1 μ g / ml , not only epithelial cells of neural tube lost and ependymocytes were out of order but urinogenital sinus and liver appeared some pathological changes .

研究即早基因（IEG）在氯化甲基汞神经毒性机制中的作用。

Objective To study the role of immediate early genes ( IEG ) in the neurotoxic mechanism of chloro methylmercury .

PKC-MAPK信号通路及PARP在长期接触低剂量氯化甲基汞所致脑发育损伤中的作用

Role of PKC-MAPK Pathway and PARP in Neurodevelopmental Toxicity by Prolonged Low Level Exposure of MMC

结果表明：氯化甲基汞的体外最小致畸剂量为0.2μg／ml，除VYS直径和VYS血管形成2项指标外，其它各指标值均明显低于对照组（P＜0.05）。

The experimental results showed that minimum teratogenic dose of methylmercury was 0.2 μ g / ml. Most parameters of the test groups were lower than that of control group ( P < 0.05 ) .

c-FOS参与了甲基汞对大鼠大脑皮层损伤毒性过程，即刻早期基因c-FOS可以作为甲基汞神经毒性检测评价效应指标。

The c-FOS has participated in the toxicity process of cerebral cortex injury by methyl mercury . The immediate early gene c-FOS can be used as an effective index of detecting and assessing neurotoxicology of methyl mercury .

目的通过体内与体外实验相结合的方法研究氯化甲基汞（MMC）对脑神经细胞CFos表达的影响，探讨甲基汞所致脑发育损伤的机制。

Objective In order to study the mechanism of injury in the brain development caused by methylmercury chloride ( MMC ) and its effects on the c-fos expression in brain nerve cells of the rats with experiments in vitro and in vivo .

结果显示，三个厂区周边土壤中总汞含量在几十到几百ng·g-1，而甲基汞的含量则很低。

The result indicated that the content of mercury in the soil around three factories was from several ten to several hundred ng · g - 1 , while that of methylmercury was very low .