伤害刺激

牙髓炎性和机械损伤性伤害刺激诱导大鼠Vc内Fos表达的比较研究

The study of the expression of Fos in Vc of rats induced by the pulpal inflammatory and mechanical noxious stimulation

[结论]缺氧预处理能够提高神经干细胞应对伤害刺激时内源性EPO的表达，并降低再次缺氧时诱导EPO表达的阈值。

[ Conclusion ] The hypoxic preconditioning can lead an increased endogenous EPO expression and decrease the threshold of EPO expression against subsequent lethal stimulation during a certain period .

除个别脑区外，神经系统在生理状态下不表达HO-1。新近发现，外周器官创伤可启动脑组织表达HO-1，提示HO-1/CO与伤害刺激信息的调制有联系。

According to the recent studies , the trauma of peripheral organs can induce the expression of HO-1 in the brain tissue , which indicated that there might be some relationship between HO-1 / CO and the modulation of injury stimulus .

机械伤害刺激激活丘脑-皮层神经网络的多通道记录研究

The activation of thalamo-cortical neural network by noxious mechanical stimulation : a multichannel recording study

结论：（1）异丙酚对内脏伤害刺激具有抑制作用；

[ Conclusions ] ( 1 ) Propofol has antinociceptive effect on visceral nociception induced by CRD .

缺氧作为一种伤害刺激能够导致机体整体直至细胞水平发生相应的反应。

As harmful stimulation , hypoxia can lead to the relevant response from the whole body level to the cell level .

本文介绍用微电极记录脊髓背角神经元对伤害刺激单位放电的反应及计算机处理方法。

The method that microelectrode is used to record unit discharge of spinal dorsal horn to respond to nocuous stimulus and its computer processing is described .

当中枢神经系统遭受到伤害刺激时，作为存在于中枢神经系统的固有免疫细胞，小胶质细胞被迅速激活并发挥双重作用。

When the central nervous system suffered damage stimulus , as the innate immune cells present in the central nervous system , microglia are rapidly activated and play a dual role .

损伤和强烈的伤害刺激能导致痛过敏。痛过敏现象的产生是由外周敏化（初级伤害感受器的敏感性增加）和中枢敏化（脊髓背角以及其他中枢神经元敏感性增加）引起的。

Injuries and intense noxious stimuli result in pain hypersensitivity , which is contributed by peripheral sensitization ( increased sensitivity of primary sensory nociceptors ) and central sensitization ( increased sensitivity of spinal dorsal horn and other CNS neurons ) .

本实验同时表明，6%酒精并没有直接增强细胞对伤害刺激的抵抗能力，而且也没有使胃粘膜的血流量和微循环得到改善。

The results also indicated that stimulus with 6 % ethanol did not increase the ability of cells to resist the injury of noxious agents ( 0.6N HC1 , HCl-ethanol and so on ) . And the blood flow and microcirculation of gastric mucosa were not improved .

P物质具有强大的血管扩张活性，其参与皮肤伤害性刺激的感受与局部血管功能的调节。

P-substance has strong vasodilative activity and is involved in the sensation of the skin to traumatic stimulation and the modulation of local vascular function .

外周P物质受体介导伤害性刺激所致脊髓背角c-fos基因的表达

Peripheral substance P receptor mediating c-fos gene expression in the spinal dorsal horn induced by peripheral noxious stimulation

胃伤害性刺激对大鼠下丘脑乳头体前区内SPmRNA阳性神经元的影响

Noxious stimulation to stomach effects on neurons containing SP mRNA in premamillary region of rat hypothalamus

后角CGRP可能与伤害性刺激传导有关。

CGRP in posterior horn may be related to noxious stimulation conduction .

内脏伤害性刺激后Fos在大鼠脑内NOS阳性神经元内的表达

Expression of Fos in NOS-positive neurons in rat brain following visceral noxious stimulation

VEGF在神经病理性疼痛大鼠脊髓的表达及其对伤害性刺激反应的易化作用

The Expressions of VEGF in the Spinal Cords of Rats with Neuropathic Pain and the Facilitating Effects on Noxious Stimuli

方法：选用SD大鼠，采用直结肠扩张（CRD）作为内脏的伤害性刺激。

Methods In SD rats CRD was used as noxious visceral stimuli .

大鼠牙髓伤害性刺激诱导延髓内Fos表达

Noxious dental pulp stimulation induced Fos expression in the medulla oblongata of the rat

大鼠腹膜伤害性刺激c-fos在中枢神经系统表达的实验研究

Empirical study of the rat peritoneal noxious stimulation inducing expression of c-fos in central nervous system

大鼠胃肠道伤害性刺激引起的中枢神经系统c-fos表达

C-fos expression within central nervous system of the rat following gastrointestinal ( visceral ) noxious stimulation

伤害性刺激电针后中缝背核生长抑素mRNA的表达及其与5-HT的共存

Expression of SOM mRNA and Coexistence of SOM mRNA and 5-HT in Nucleus Raphe Dorsalis Following Noxious Stimulation and Electroacupuncture Analgesia

鼠尾伤害性刺激与颧髎穴电针后弓状核-导水管周围灰质-延髓头端腹内侧区的前脑啡肽原mRNA的表达及与Fos蛋白共存

Expression of preproenkephalin mRNA and its coexistence with FOS in rat ar-pag-rvm following tail nociceptive stimulation and acupuncture in Quanliao point

结果（1）伤害性刺激使大鼠丘脑PF的PEN诱发放电频率增加；

Results ( 1 ) When the noxious stimuli were administered , the frequency of evoked discharge of PEN was increased ;

胃伤害性刺激诱导大鼠脑干星形胶质细胞GFAP蛋白表达及其与神经元的关系

Expression of GFAP in rat brain stem astrocytes induced by stomachic nociception and relationship to neurons

吗啡－3－葡糖苷酸（M3G）对伤害性刺激诱导大鼠脊髓c－fos表达的影响

Morphine-3-glucuronide ( m3g ) potentiates noxious stimulus-evoked FOS protein-like immunoreactivity in the rat spinal cord

胃肠道伤害性刺激诱导中缝背核触液神经元Fos表达

Gastrointestinal noxious stimulation induced expression of Fos protein in the cerebrospinal fluid contacting neurons of dorsal raphe nucleus

大鼠脑干内向杏仁中央核投射的神经元对胃肠道伤害性刺激的FOS表达

The gastrointestinal noxious stimulation induced Fos expression of brainstem neurons projecting to the central amygdaloid nucleus in the rat

外周伤害性刺激后颈髓和三叉神经脊束核尾侧亚核中向中缝背核投射神经元的c-fos表达

Peripheral noxious stimulation-induced c-fos expression in neurons of cervical cord and caudal spinal trigeminal nucleus projection to the dorsal raphe nucleus

吗啡对非伤害性刺激后CCI大鼠脊髓c-fos表达的影响

Effect of morphine on expression of c-fos induced by innoxious touch-stimulus in the spinal cord in CCI rat

结论：GIRI的伤害性刺激影响了PVN、NTS的功能。

Conclusion : The function of PVN and NTS could be affected by the GI / RI noxious stimulation .