血管性水肿

拉西地平致急性荨麻疹伴血管性水肿

Lacidipine-induced acute urticaria and angioedema

牙科治疗后面颈部软组织气肿：2例报道并强调与血管性水肿相鉴别

Soft tissue cervicofacial emphysema after dental treatment : Report of 2 cases with emphasis on the differential diagnosis of angioedema

C1抑制物的遗传缺陷是遗传性血管性水肿（HAE）的基本特征。

Hereditary angioedema ( HAE ) is characterized by an inherited defect of C1 inhibitor ( C1INH ) .

慢性荨麻疹（CU）是一类常见的皮肤病，表现为反复出现的红斑、风团伴瘙痒（可合并血管性水肿），持续至少六周以上。

Chronic urticaria is a common skin disease , manifested by recurrent erythema , wheal with itching ( may be combined with angioedema ), sustained at least six weeks .

遗传性血管性水肿是由C1酯酶抑制剂基因突变所致的一种罕见的常染色体显性遗传性疾病。

Hereditary angioedema is a rare autosomal dominant genodermatosis caused by C1 inhibitor ( C1 INH ) gene mutations .

遗传性血管性水肿是一种由于C1酯酶抑制剂的合成障碍或功能缺陷所致的常染色体显性遗传病。

Hereditary angioedema ( HAE ) is an autosomal dominant genetic disease caused by synthesis defect or dysfunction of C1 esterase inhibitor .

结论：PRES的血管性水肿主要累及后循环区域，但前循环也常有受累。

CONCLUSION : Vasogenic edema in PRES involves predominantly the posterior circulation territories , but anterior circulation structures are also frequently involved .

血管性水肿的历史与其他DPP-4抑制剂。

History of angioedema with other DPP-4 inhibitors .

以突发突止腹痛、腹水为表现的神经血管性水肿1例报告

One case report of nerve angioedema associated with transient abdominal pain and ascites

急性缺血性卒中患者溶栓期出现危及生命的口舌部血管性水肿

Life-threatening orolingual angioedema during thrombolysis in acute ischemic stroke

遗传性血管性水肿分子遗传学进展

Molecular Genetics Progress of Hereditary Angioedema

对遗传性血管性水肿的病因及发病机制、临床表现及分型、治疗进行概述。

The etiology , pathogenesis , clinical manifestations , as well as treatment of the disease are summarized .

发疹型药疹最为常见，其次为固定性红斑和荨麻疹与血管性水肿。

It showed that the most common form among them was exanthematous type , secondly were fixed eruption and urticaria angioedema types .

最初终点为每周期血管性水肿发作数，由受试者自行控制。

The primary end point was the number of attacks of angioedema per period , with each subject acting as his or her own control .

另外一位过敏患者表现为荨麻疹，血管性水肿，喉头水肿和心悸，第三位女孩则是声音嘶哑。

Another case was considered to be anaphylaxis with symptoms of urticaria , angioedema , laryngeal edema , and palpitations , and the third case was considered to be a hoarse voice .

大鼠急性脑缺血再灌注后血管源性水肿MRI与脑组织VEGF表达

MRI manifestations of vasogenic brain edema in acute ischemia reperfusion rats and VEGF expression of ischemic brain tissue

DWI和ADC图联合应用可鉴别血管源性水肿和细胞毒性水肿。

DWI and ADC map can be combined with identification of vasogenic edema and cytotoxic edema . 5 .

显微病理学见病灶以血管源性水肿为主，12h后出现静脉性脑梗死、脑出血。

The microscopic changes were mainly vasogenic edema and followed by venous hemorrhagic infarction in 12 h.

3例患者在停止治疗后几小时内，血管神经性水肿均恢复。

The angioneurotic oedema resolved in all 3 patients within hours of discontinuing treatment with the agent .

结论掌握了颌面部慢性血管神经性水肿的临床特征后，该病诊断容易，治疗效果满意。

Conclusion Diagnoses was easy for this disease when its clinical manifestations were found out and the treatment was effective .

其机制可能：（1）内毒素可直接损害血脑屏障，产生血管源性水肿；

The possible mechanisms are : 1 . Endotoxin may cause the damage of blood brain barrier directly , and thus results in angio-edema .

HE染色光镜和透射电子显微镜观察，3小时开始即出现广泛水肿表现，同时存在细胞毒性水肿和血管源性水肿，三者形态学表现相符。

The cytotoxic brain edema and vasogenic brain edema existed from 3 hour to 24 hour showed by the results of HE light microscope and transmissional electron microscope .

大鼠急性期缺血再灌注血管原性脑水肿MRI表现及与血管内皮细胞明胶酶B表达的关系

MRI manifestations of vasogenic brain edema of acute ischemia / reperfusion of rats and the relationship between it and gelatinase B expression of vascular endothelium

实验性大鼠急性期脑缺血血脑屏障开放、血管源性脑水肿MRI与脑组织MMP-9、VEGF表达

Blood-brain Barrier Opening and Vasogenic Brain Edema of Experimental Acute Ischemia of Rats : MRI Evaluation , and the Correlation with MMP-9 and VEGF Expression in Infarcted Brain Tissue

其中血管源性脑水肿发生以血脑屏障（BBB）通透性的改变为主要原因。

The major cause of vasogenic cerebral edema is the change of blood-brain barrier ( BBB ) permeability .

血管源性脑水肿（vasogenicbrainedema，VBE）是脑缺血后的重要病理生理变化，可加重因缺血缺氧而引起的神经细胞损伤。

Vasogenic brain edema ( VBE ) is very important pathological change after cerebral ischemia , which can aggravate the ischemic damage .

方法测定MRIT1和T2值及水肿区大小，观察甘露醇用药前后对脑血管病性脑水肿的治疗作用。

Methods The changes of proton relaxation times ( T1 and T2 ) and brain edema sizes in MRI were studied in patients with brain edema of acute cerebral vascular diseases before and after intravenously infused with mannitol .

【结论】外伤后2hNF能加重血管源性脑水肿，但能改善细胞毒性脑水肿，而在24h能改善血管源性脑水肿。

[ Conclusion ] NF could aggravate the vasogenic brain edema but alleviate cytotoxic brain edema at 2 hr after insult and could alleviate cytogenous brain edema at 24 hr .

酶屏障抑制剂951治疗血管源性脑水肿

Treatment of Vasogenic Brain Edema with Inhibitor 951 of Enzyme Barrier

碱性磷酸酶单克隆抗体对大鼠血管源性脑水肿的治疗作用

Therapeutic role on monoclonal antibody of alkaline phosphatase to vasogenic cerebral edema in rats